Saturday, April 30, 2011

Shortness Heightens Heart Risk

Today I just go to my bookshelf to look for some book that I can read to spend my free time. Guess what? Reader's Digest book! Although it is August 2010, it's okay since reading that book is very interesting no matter how many times you read it. It just like you buy a new book every times you read it. After some enjoying my time reading it, my eyes caught an attention to the Health Smart section,Shortness Heightens Heart Risk.
So, I want to share this with all my blog readers:
Short people have a higher risk of heart disease. Finish researchers suggested in the European Heart Journal that women under 1.53m and men shorter than 1.65m are 1.5 times more likely to develop and die from heart disease.
Studies showed that there is a strong link between stature and heart disease risk, although the reason is not conclusively clear. It could be that shorter people have shorter blood vessels to the heart, whch may make it easier to clog. But researchers say that short people should not worry unduly. There are also other factors like weight, lifestyle habits such as smoking, drinking and exercise that contribute to heart risk.
This does not mean that tall people are protected against heart disease, only that short adults should pay attention and realize their increased risk.
What's most important for maintaining a healthy heart is to exercise regularly, eat a healthy and balanced diet, stop smoking and watch your weight.

I also found that, they also have the post in their website: Instead of reading about that post, we may also get a knowledge about other things on health. Hopefully you enjoy it!

Thursday, April 28, 2011

Ovarian cancer starts elsewhere

Recently I have do some searching about ovarian cancer. Then, I found that this post caught my interest. Hopefully, this share help us too get better understanding about ovary cancer. Don't forget to take care of your health.

By AFP Relax news:
A new finding could provide clues on how to attack ovarian cancer, which often causes no early symptoms.

(WASHINGTON-AFP) - US researchers have recreated the process by which ovarian cancer forms in the lab, providing solid evidence that the tumors start in the fallopian tubes, not the ovaries, a study said. The finding could provide clues on how to attack ovarian cancer, which often causes no early symptoms and by the time it is found has spread so much that the tumors are impossible to stop.

Ovarian cancer is the fifth deadliest cancer among women, affecting 200,000 women worldwide annually and killing 115,000 women on average each year.

Several studies have theorized that the cancer may originate elsewhere, but the latest research by scientists at the Dana-Farber Cancer Institute in Boston shows how the cancer takes root first in fallopian tissue.

The fallopian tubes are the pathways by which a woman's egg travels from the ovary to the uterus as part of her reproductive cycle.

Ronny Drapkin, senior author of the study in the Proceedings of the National Academy of Sciences, said previous examinations of fallopian tissue taken from women genetically predisposed to ovarian cancer have shown "patches of cells that were predecessors of serious cancers."

So they decided to try and replicate the process of cancer formation in the lab.Researchers took fallopian cells and altered their genetic programming so they would divide much like cancer cells."Like true tumor cells, these 'artificial' cancer cells proliferated rapidly and were able to leave their home tissue and grow elsewhere," said the study.

"When implanted in laboratory animals, they also gave rise to tumors that were structurally, behaviorally, and genomically similar to human HGSOC (high-grade serious ovarian cancer)."Drapkin said the findings demonstrate that fallopian cells are the source of ovarian cancer, and offer clues for future treatment.

"Such studies will help us identify different types of high-grade serous ovarian cancer, as well as possibly discover biomarkers -- proteins in the blood -- that signal the presence of the disease," said Drapkin, an assistant professor at Harvard Medical School."Ultimately, the model will enable us to test potential therapies to determine which work best in each type of the disease."

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